Accelerated cellular senescence in solid tumor therapy

Accelerated cellular senescence (ACS) is an emerging concept that implicates sustained, telomere-independent cell cycle arrest of neoplastic cells in response to chemotherapeutic agents, ionizing radiation, oxidative stress, or the presence of selective oncogenic stimuli. Recent evidence suggests th...

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Veröffentlicht in:Experimental Oncology
Datum:2012
Hauptverfasser: Wu, P.C., Wang, Q., Grobman, L., Chu, E., Wu, D.Y.
Format: Artikel
Sprache:English
Veröffentlicht: Інститут експериментальної патології, онкології і радіобіології ім. Р.Є. Кавецького НАН України 2012
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Online Zugang:https://nasplib.isofts.kiev.ua/handle/123456789/139056
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Назва журналу:Digital Library of Periodicals of National Academy of Sciences of Ukraine
Zitieren:Accelerated cellular senescence in solid tumor therapy / P.C. Wu, Q. Wang, L. Grobman, E. Chu, D.Y. Wu // Experimental Oncology. — 2012. — Т. 34, № 3. — С. 298-305. — Бібліогр.: 85 назв. — англ.

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Digital Library of Periodicals of National Academy of Sciences of Ukraine
id nasplib_isofts_kiev_ua-123456789-139056
record_format dspace
spelling Wu, P.C.
Wang, Q.
Grobman, L.
Chu, E.
Wu, D.Y.
2018-06-19T18:53:53Z
2018-06-19T18:53:53Z
2012
Accelerated cellular senescence in solid tumor therapy / P.C. Wu, Q. Wang, L. Grobman, E. Chu, D.Y. Wu // Experimental Oncology. — 2012. — Т. 34, № 3. — С. 298-305. — Бібліогр.: 85 назв. — англ.
1812-9269
https://nasplib.isofts.kiev.ua/handle/123456789/139056
Accelerated cellular senescence (ACS) is an emerging concept that implicates sustained, telomere-independent cell cycle arrest of neoplastic cells in response to chemotherapeutic agents, ionizing radiation, oxidative stress, or the presence of selective oncogenic stimuli. Recent evidence suggests that a subset of tumor cells induced in a state of reversible ACS can escape cell cycle arrest and resume proliferation accounting for cancer progression. The purpose of this review is to describe our current understanding of ACS including signaling pathways of senescence escape, role of senescence biomarkers, and rationale for senescence-based therapy. This article is part of a Special Issue entitled “Apoptosis: Four Decades Later”.
en
Інститут експериментальної патології, онкології і радіобіології ім. Р.Є. Кавецького НАН України
Experimental Oncology
Reviews
Accelerated cellular senescence in solid tumor therapy
Article
published earlier
institution Digital Library of Periodicals of National Academy of Sciences of Ukraine
collection DSpace DC
title Accelerated cellular senescence in solid tumor therapy
spellingShingle Accelerated cellular senescence in solid tumor therapy
Wu, P.C.
Wang, Q.
Grobman, L.
Chu, E.
Wu, D.Y.
Reviews
title_short Accelerated cellular senescence in solid tumor therapy
title_full Accelerated cellular senescence in solid tumor therapy
title_fullStr Accelerated cellular senescence in solid tumor therapy
title_full_unstemmed Accelerated cellular senescence in solid tumor therapy
title_sort accelerated cellular senescence in solid tumor therapy
author Wu, P.C.
Wang, Q.
Grobman, L.
Chu, E.
Wu, D.Y.
author_facet Wu, P.C.
Wang, Q.
Grobman, L.
Chu, E.
Wu, D.Y.
topic Reviews
topic_facet Reviews
publishDate 2012
language English
container_title Experimental Oncology
publisher Інститут експериментальної патології, онкології і радіобіології ім. Р.Є. Кавецького НАН України
format Article
description Accelerated cellular senescence (ACS) is an emerging concept that implicates sustained, telomere-independent cell cycle arrest of neoplastic cells in response to chemotherapeutic agents, ionizing radiation, oxidative stress, or the presence of selective oncogenic stimuli. Recent evidence suggests that a subset of tumor cells induced in a state of reversible ACS can escape cell cycle arrest and resume proliferation accounting for cancer progression. The purpose of this review is to describe our current understanding of ACS including signaling pathways of senescence escape, role of senescence biomarkers, and rationale for senescence-based therapy. This article is part of a Special Issue entitled “Apoptosis: Four Decades Later”.
issn 1812-9269
url https://nasplib.isofts.kiev.ua/handle/123456789/139056
citation_txt Accelerated cellular senescence in solid tumor therapy / P.C. Wu, Q. Wang, L. Grobman, E. Chu, D.Y. Wu // Experimental Oncology. — 2012. — Т. 34, № 3. — С. 298-305. — Бібліогр.: 85 назв. — англ.
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first_indexed 2025-11-30T19:03:30Z
last_indexed 2025-11-30T19:03:30Z
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