Polymorphism of DNA mismatch repair genes in endometrial cancer

Endometrial cancer (EC) is the second most common malignancy associated with hereditary non-polyposis colorectal cancer (HNPCC) family. The development of HNPCC is associated with defects in DNA mismatch repair (MMR) pathway resulting in microsatellite instability (MSI). MSI is present in a greater...

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Veröffentlicht in:Experimental Oncology
Datum:2015
Hauptverfasser: Poplawski, T., Sobczuk, A., Sarnik, J., Pawlowska, E., Blasiak, J.
Format: Artikel
Sprache:English
Veröffentlicht: Інститут експериментальної патології, онкології і радіобіології ім. Р.Є. Кавецького НАН України 2015
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Online Zugang:https://nasplib.isofts.kiev.ua/handle/123456789/145454
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Zitieren:Polymorphism of DNA mismatch repair genes in endometrial cancer / T. Poplawski, A. Sobczuk, J. Sarnik, E. Pawlowska, J. Blasiak // Experimental Oncology. — 2015. — Т. 37, № 1. — С. 44-47. — Бібліогр.: 29 назв. — англ.

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Digital Library of Periodicals of National Academy of Sciences of Ukraine
id nasplib_isofts_kiev_ua-123456789-145454
record_format dspace
spelling Poplawski, T.
Sobczuk, A.
Sarnik, J.
Pawlowska, E.
Blasiak, J.
2019-01-21T21:26:54Z
2019-01-21T21:26:54Z
2015
Polymorphism of DNA mismatch repair genes in endometrial cancer / T. Poplawski, A. Sobczuk, J. Sarnik, E. Pawlowska, J. Blasiak // Experimental Oncology. — 2015. — Т. 37, № 1. — С. 44-47. — Бібліогр.: 29 назв. — англ.
1812-9269
https://nasplib.isofts.kiev.ua/handle/123456789/145454
Endometrial cancer (EC) is the second most common malignancy associated with hereditary non-polyposis colorectal cancer (HNPCC) family. The development of HNPCC is associated with defects in DNA mismatch repair (MMR) pathway resulting in microsatellite instability (MSI). MSI is present in a greater number of EC than can be accounted for by inherited MMR mutations, therefore alternative mechanisms may underline defective MMR in EC, including polymorphic variation. Aim: We checked the association between EC occurrence and two polymorphisms of MMR genes: a 1032G>A (rs4987188) transition in the hMSH2 gene resulting in a Gly22Asp substitution and a –93G>A (rs1800734) transition in the promoter of the hMLH1 gene. Material and methods: These polymorphisms were genotyped in DNA from peripheral blood lymphocytes of 100 EC patients and 100 age-matched women by restriction fragment length polymorphism PCR. Results: A positive association (OR 4.18; 95% CI 2.23–7.84) was found for the G/A genotype of the –93G>A polymorphism of the hMLH1 gene and EC occurrence. On the ot­her hand, the A allele of this polymorphism was associated with decreased EC occurrence. The Gly/Gly genotype slightly increased the effect of the –93G>A-G/A genotype (OR 4.52; CI 2.41–8.49). Our results suggest that the –93G>A polymorphism of the hMLH1 gene singly and in combination with the Gly322Asp polymorphism of the hMSH2 gene may increase the risk of EC. Key Words: hMSH2, hMLH1, endometrial cancer, genetic polymorphism, MMR.
en
Інститут експериментальної патології, онкології і радіобіології ім. Р.Є. Кавецького НАН України
Experimental Oncology
Original contributions
Polymorphism of DNA mismatch repair genes in endometrial cancer
Article
published earlier
institution Digital Library of Periodicals of National Academy of Sciences of Ukraine
collection DSpace DC
title Polymorphism of DNA mismatch repair genes in endometrial cancer
spellingShingle Polymorphism of DNA mismatch repair genes in endometrial cancer
Poplawski, T.
Sobczuk, A.
Sarnik, J.
Pawlowska, E.
Blasiak, J.
Original contributions
title_short Polymorphism of DNA mismatch repair genes in endometrial cancer
title_full Polymorphism of DNA mismatch repair genes in endometrial cancer
title_fullStr Polymorphism of DNA mismatch repair genes in endometrial cancer
title_full_unstemmed Polymorphism of DNA mismatch repair genes in endometrial cancer
title_sort polymorphism of dna mismatch repair genes in endometrial cancer
author Poplawski, T.
Sobczuk, A.
Sarnik, J.
Pawlowska, E.
Blasiak, J.
author_facet Poplawski, T.
Sobczuk, A.
Sarnik, J.
Pawlowska, E.
Blasiak, J.
topic Original contributions
topic_facet Original contributions
publishDate 2015
language English
container_title Experimental Oncology
publisher Інститут експериментальної патології, онкології і радіобіології ім. Р.Є. Кавецького НАН України
format Article
description Endometrial cancer (EC) is the second most common malignancy associated with hereditary non-polyposis colorectal cancer (HNPCC) family. The development of HNPCC is associated with defects in DNA mismatch repair (MMR) pathway resulting in microsatellite instability (MSI). MSI is present in a greater number of EC than can be accounted for by inherited MMR mutations, therefore alternative mechanisms may underline defective MMR in EC, including polymorphic variation. Aim: We checked the association between EC occurrence and two polymorphisms of MMR genes: a 1032G>A (rs4987188) transition in the hMSH2 gene resulting in a Gly22Asp substitution and a –93G>A (rs1800734) transition in the promoter of the hMLH1 gene. Material and methods: These polymorphisms were genotyped in DNA from peripheral blood lymphocytes of 100 EC patients and 100 age-matched women by restriction fragment length polymorphism PCR. Results: A positive association (OR 4.18; 95% CI 2.23–7.84) was found for the G/A genotype of the –93G>A polymorphism of the hMLH1 gene and EC occurrence. On the ot­her hand, the A allele of this polymorphism was associated with decreased EC occurrence. The Gly/Gly genotype slightly increased the effect of the –93G>A-G/A genotype (OR 4.52; CI 2.41–8.49). Our results suggest that the –93G>A polymorphism of the hMLH1 gene singly and in combination with the Gly322Asp polymorphism of the hMSH2 gene may increase the risk of EC. Key Words: hMSH2, hMLH1, endometrial cancer, genetic polymorphism, MMR.
issn 1812-9269
url https://nasplib.isofts.kiev.ua/handle/123456789/145454
citation_txt Polymorphism of DNA mismatch repair genes in endometrial cancer / T. Poplawski, A. Sobczuk, J. Sarnik, E. Pawlowska, J. Blasiak // Experimental Oncology. — 2015. — Т. 37, № 1. — С. 44-47. — Бібліогр.: 29 назв. — англ.
work_keys_str_mv AT poplawskit polymorphismofdnamismatchrepairgenesinendometrialcancer
AT sobczuka polymorphismofdnamismatchrepairgenesinendometrialcancer
AT sarnikj polymorphismofdnamismatchrepairgenesinendometrialcancer
AT pawlowskae polymorphismofdnamismatchrepairgenesinendometrialcancer
AT blasiakj polymorphismofdnamismatchrepairgenesinendometrialcancer
first_indexed 2025-12-07T16:32:40Z
last_indexed 2025-12-07T16:32:40Z
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